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Thus, high levels of ROS present in tumor cells could result from either an amplified metabolic activity, mitochondrial dysfunction, peroxisome activity, deregulated cell receptor signaling, oncogene activity, enhanced activity of cyclooxygenase, lysyl oxidase, and thymidine phosphorylase, or communication with the immune infiltrate (Babior, 2005), suggesting that ROS and RNS can support numerous facets of tumor development and progression

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L-DOPA alone contributed to strong recovery of motion phenotypes during initial phase, however, its effect lagged in aged flies which showed movement defects similar to untreated park 13 mutants (Fig