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Originally engineered at Washington State University as a blood-brain barrier permeable procognitive agent, Dihexa binds with ultra-high affinity to Hepatocyte Growth Factor (HGF)
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OS extract leads to a reduction in intracellular calcium levels and subsequent relaxation via ion influx inhibition through VOCC, as the nifedipine action, and reduces vasoconstriction caused by phenylephrine, demonstrating its involvement in IP3 and/or ryanodine receptors
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