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2023;54:S23–S28
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Considering the evidence for consumption (and loss) of ascorbate in the ER due to 2-ODD activity, and the large demand for hydroxylated extracellular proteins such as extensin and AGPs during cell expansion, it is likely that growth arrest in ascorbate null mutants could be caused by limited 2-ODD activity while the 80–90% decrease in ascorbate in the vtc mutants is still sufficient to support 2-ODD activity
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