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GHK-Cu is associated with antioxidant and anti-inflammatory activity, making it relevant in studies related to oxidative stress and cellular protection

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Depletion of GSH by BSO, an irreversible inhibitor of GCL , that does not by itself induce nigrostriatal damage in vivo [80], potentiates the amount of MPTP–induced tyrosine hydroxylase-positive (TH + ) neuron death in the SNpc (48.6% cell death compared to 30.1% cell death) [69, 80]