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However, increased flux through glycolysis, which is commonly seen under diabetic conditions, results in glycolytic overload, which leads to an abnormal increase in the concentrations of glycolytic intermediates that can be shunted into different damaging pathways such as polyol, hexosamine, protein kinase C (PKC) pathways, and advanced glycation end products (AGEs) as shown in Figure 2 (Yumnamcha et al., 2020)
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[DOI] [PubMed] [Google Scholar] Wall TL, Peterson CM, Peterson KP, et al
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