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Nonetheless, there is currently no sufficient information concerning drug or chemical distribution within tissues based on such data, highlighting the need for further research on this issue [18,19,20,21,22]

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30 min pretreatment, 24 h AβO exposure) dose-dependently inhibits the release of proinflammatory mediators (TNF-α, IL-1β, NO) and the expression of proinflammatory proteins (iNOS, COX-2) induced by AβO in BV-2 cells, suppresses AβO-induced oxidative stress, and restores the antioxidant capacity of cells [5]

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