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PPP is the major source of NADPH in the cell, and the increase in the glucose flux away from glycolysis to the PPP, as well as the transcriptional activation of PPP genes, such as G6PD, PGD, TKT, and TALDO, which are NRF2 targets observed by SF only under HG conditions, will lead to an increase in NADPH pool that supports antioxidant reactions [25]

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