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Knockout of the de novo DNA methyltransferase DNMT3A in human induced pluripotent stem cell (iPSC)-derived cardiomyocytes within engineered heart tissues (EHTs) causes mitochondrial dysfunction, impaired glucose metabolism, and intracellular lipid accumulation under baseline conditions
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MOTS-c Before You Start: The Critical Protocol Facts MOTS-c is a mitochondria-derived peptide that regulates cellular energy metabolism, which makes it mechanistically distinct from every other peptide in a standard protocol library
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