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Structural analyses show that cagrilintide mimics amylin’s receptor engagement while introducing conformational differences at select positions, such as Pro37, which enhances CTR interaction and modulates receptor dynamics (Cao et al

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ROS accumulation triggers OS, cellular damage, mitochondrial dysfunction, Aβ deposition, Tau hyperphosphorylation, synaptic loss, and neuronal death—key factors in AD progression (Aliev et al., 2004