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intracellular half-life is prolonged because of lysosomal release and cytosolic resequestration, creating a depot effect that supports monthly maintenance dosing for chronic deficiencies
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The different expression levels observed in APP/PS1 animals, especially for the m- Gpx4 isoform, suggest a genotype-related transcriptional regulation, which fits well with the increased levels of hippocampal oxidative stress demonstrated in transgenic animals (Aso et al., 2012)
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