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suggested that inflammatory injury in the intestine might trigger the observed increase in plasma GPX activity, which is associated with increased GPX3 mRNA levels in the kidney [165]
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The pathology of AD is characterized by the accumulation of extracellular amyloid β (Aβ) plaques and the formation of intracellular neurofibrillary tangles composed of hyperphosphorylated tau protein (38)
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