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Conclusion ATR and WEE1 inhibitors have produced credible clinical activity in selected DNA damage response-defined cancers, including responses after PARP inhibitor resistance in ovarian cancer.[1-3] The evidence remains insufficient to establish post-PARP disease as a single biologic entity or to support routine use of an ATR- or WEE1-directed regimen outside an appropriate clinical trial
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Size:3.33 oz.
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